Blindness is an injury to the retina, the part of the eye that is used to see colors and see through objects.
It’s caused by damage to the optic nerve, which connects the retina to the brain.
Studies suggest that the damage to this nerve may cause damage to vision.
For the first time, researchers have found evidence that a genetic mutation that makes a protein called SERT1 in the retina produces defects that prevent blindness from occurring.
They also found that a mutation in the gene that controls SERT2, which regulates the production of the protein, could explain the link between SERT and blindness.
The findings are published online today (Jan. 5) in the journal Nature.
The study looked at a group of blind patients with degenerative eye diseases.
They were given a test called the Spot-Tests to determine if their blindness was caused by their genetic mutation or a more common disorder called refractive error.
A second test, called the Blindness Questionnaire, assesses the severity of the patients’ blindness.
Blindness was diagnosed using the Spot Tests, which are a standard procedure to evaluate patients with eye disease.
The researchers used a method that combines the Spot tests and the BlindNESS Questionnaire to determine the genetic mutations.
The mutation that causes blindness is located in the region of SERT called the retinal pigment epithelial protein, or RPEP.
This protein is involved in vision.
This is important because the damage that the gene mutation causes in the retina can damage the vision in the eyes of some people.
Previous studies have shown that SERT is required for the protein to function.
This gene mutation is one of several that cause the protein’s destruction, but it’s not known why.
The scientists found that the mutations that make SERT in the eye occur in a region called the RPE1B region.
This region is involved with nerve cell function and is involved both in producing nerve cells and in producing the proteins that are required for nerve cell activity.
The RPE protein has been known to cause vision defects in people with degeneration of the retinas.
SERT gene mutations that cause blindness The scientists discovered that the SERT protein was mutated in a specific region of the retina called the RPEP region.
The gene mutation that caused the blindness was found in the RPep region of that gene.
The protein was disrupted in the RPP1B area of the RPEp, which is part of a nerve cell called the nerve root.
The nerve root is the area of nerve tissue that connects the optic nerves to the rest of the brain and spinal cord.
The disruption of the nerve cell produces the same type of damage that occurs in the visual system.
Blind people with vision loss in the sighted eye have an increased risk of developing retinal nerve disease and, therefore, blindness in the blind.
The genes responsible for this defect are found in more than 200 different families, so it’s difficult to pinpoint a specific gene mutation.
The genetic damage is also more likely to occur in people of Asian descent, who have a lower rate of mutations.
Understanding how the protein plays a role in vision is important for treating vision loss and treating retinal disease.
It could also be used to develop a better eye sight test.
“This is one step toward improving the diagnosis and treatment of vision loss due to retinal degeneration,” said study co-author Dr. Robert E. Krasner, an associate professor of ophthalmology at the University of Washington.
“Our next steps are to investigate the function of the gene in the other eye and determine how it is expressed in the brain, to see if it might be able to repair the damage in the optic system.”
More research is needed to understand how the RPP protein and the protein that regulates it are involved in different types of vision disorders.
Understanding more about the RPPs role in the vision system could help doctors diagnose these conditions more accurately.
The SERT mutations have been known for some time.
Researchers have used other methods to study the proteins, but the mutation that led to blindness was the first to be identified.
This could be one of the reasons that researchers have not been able to find an accurate diagnosis of these conditions.
The team plans to continue studying the protein and other genetic changes that affect vision.
Researchers also plan to explore the gene mutations in a new group of people.
The next steps for this group include studying how the mutations affect the retina in people who have already developed vision loss.
They will also examine how the mutation affects the function and repair of the vision cells in the diseased eye.